Maybe Your Prescriptive Medications Are Making You Sick!!

Based upon recent research the medications that you have been taking with such great confidence may be irreversibly damaging your health.

Mitochondrial dysfunction is to blame for many toxicities caused by drugs and may explain the side effects of many medications.

So what is a mitochondria?

Why is it so important?

The mitochondria are present in both animal and plan a sales they are rod-shaped structures that are in close with into membranes–the outer membrane in the inner membrane. The membranes are made up of phospholipids and proteins. The mitochondria are called the “powerhouse of the cell”. They contain a number of in signs and proteins that help process carbohydrates and fats obtained from the food we eat to relieve his energy.

Be it the beating of the heart or moving of your hands, every action requires energy. The energy is still ordered in ATP (adenosine triphosphate) molecules produced in the mitochondria by the process of oxidative phosphorylation. Though there primary function is reduced energy, they also played an important role in the metabolism and symphysis of certain other substances in the body. When this small albeit important organelle, it can lead to serious health problems.

Over 15 a.m. people in the United States have chronic degenerative disorders that involve mitochondrial dysfunction. Dysfunctions of the mitochondria can affect the production of cell-specific products that are essential for proper cell functioning and energy production. This can eventually lead to cell death and failure of the organ system it can even prove to be fatal in some cases.
Dysfunction of the mitochondria can result in reduced production of energy which can lead to dysfunction of the brain, vision problems, weak muscles, restricted movement of limbs, etc. Mitochondrial disease can destroy the help of any system or any organ of the body. It can destroy heart health and digestive health. Mitochondrial dysfunction can produce symptoms such as reoccurring infections due to a weak immune system, reduce capacity of the heart, strokes, seizures, muscle fatigue, gastrointestinal problems, liver problems, diabetes, obesity, blindness and deafness studies show that mitochondrial dysfunction is the root cause of many common illnesses such as Alzheimer’s disease, Parkinson’s disease, diabetes, hypertension, heart disease, osteoporosis, cancer, autoimmune diseases like multiple sclerosis, lupus and rheumatoid arthritis, etc. Mitochondrial dysfunction plays a significant role in premature aging symptoms.

Drugs and the Mitochondria.
Mitochondrial dysfunction is to blame for many toxicities caused by drugs and may explain the side effects of many medications.

Drug skin damage mitochondria in 2 ways: Directly or indirectly. Medications can indirectly inhibit DNA transcription to electron transport chain complexes. Through these mechanisms, medications damage electron transport chain components as well as block enzymes required for normal mitochondrial function. Indirect mitochondrial damage caused by medications occurs to the production of free radicals, causing a decrease in in the genesis antioxidants such as glutathione. Medications may also deplete levels of nutrients needed for creating or proper functioning of mitochondrial enzymes are electron transport chain complexes.

Medications Documented to Cause Mitochondrial Damage
1.Alcoholism Medications
Antabusem

2.Analgesic (for Pain) and Anti-Inflammatories
Aspirin, Tylenol,Voltaren,Voltorol,Difen,Cataflam, Indocin Naprosyn, Aleve

3.Anesthetics
Bupivcaine, lidocaine, propofol

4.Angina Medications
Perhexiline, aminofdarone, DEAEH.

5.Anti-arrhythmic
Cordarone

6.Antibiotics
Tetracycline, antimycin A

7.Antidepressants
Amitriptyline, amoxapine, citalopram, Cipramil, Fluoxetine, Prozac, Symbyax, Sarafem, Foxetin, Ladose,Fluctin, Fudac, Oxetin, Seronil, Lovan.

8.Antipsychotics
Chlorpromazine, Fluphenazine, haloperidol, risperidone, clozapine, olanzapine

9.Anxiety Medications
Xanax, diazepam, Valium, Diastat

10.Barbiturates
Amobarbital, Amytal, Aprobarbital, butabarbital, Fiorinal, Sombulex, methylphenobarbital, Nembutal, phenobarbital, pentobarbital.

11.Cholesterol Medications
Statins, Lipitor, Torvast, lovastatin, Lipitor, Crestor, Zocor, Lipex, WelChol, Questran, Colestid, Pitava.

12.Cancer Medications
Mitomycin-C, ABVD, CHOP, FAC, profiromycin,doxorubicin,

13.Dementia
Tarcine,Cognex,Reminyl

14.Diabetes Medications
Metformin, Fortamet, Glucophage, Glucophage XR, Riomet,troglitazone, buformin, rosiglitazone.

15.Epilepsy/Seizure Medication
Valproic acid, Depacon, Depakene, Depakote, divalproex sodium

16.Mood Stabilizers
Lithium

17.Parkinson’s Disease Medications
Talcapone, Tasmar,Entacapone COMTan, also in the combination drug Staevo.

It is recommended to any patient who is taking medications known to cause mitochondrial dysfunction that he or she should begin a supplement regimen as soon as possible.

The supplement regimen should include:
Coenzyme Q 10
Quercetin
Nicotinamide ribosie
Acetyl L carnitine
Alpha lipoic-acid
Glucosamine
N-acetyl cysteine
Creatine monohydrate
Omega-3 fatty acids
Green tea
Resveratrol

I have incorporated more than ever the concept of fueling the cells throughout the body to ensure that they have sufficient ATP production to allow the cells performed their prime function. Specifically, when I work with patients who have chronic disease, I worked to remove the obstacles to resolution of the now self-perpetuating condition. I share with my patients the analogy of removing the proverbial splinter that has festered and thus disturbing health–sustaining homeostasis.

The mitochondria are critical to maintaining homeostasis and any malfunction of mitochondrial activity could result in disease. Implementing a mitochondrial–supporting regimen that includes exercising and daily nutrients can yield excellent results in helping patients to achieve the health goals.

Dr. G Phillip Paulk DC

I would like to thank PROTHERA, INC., Practitioner Newsletter of September 2014 for providing a significant portion of the data for this article. Citations of supporting articles can be found in the September edition.

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